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Showing 1 to 12 of 258 entries
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Liver cirrhosis-effect on QT interval and cardiac autonomic nervous system activity.

World journal of gastrointestinal pathophysiology

Tsiompanidis E, Siakavellas SI, Tentolouris A, Eleftheriadou I, Chorepsima S, Manolakis A, Oikonomou K, Tentolouris N.
PMID: 29487764
World J Gastrointest Pathophysiol. 2018 Feb 15;9(1):28-36. doi: 10.4291/wjgp.v9.i1.28.

AIM: To examine the impact of liver cirrhosis on QT interval and cardiac autonomic neuropathy (CAN).METHODS: A total of 51 patients with cirrhosis and 51 controls were examined. Standard 12-lead electrocardiogram recordings were obtained and QT as well as...

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Tsiompanidis E, Siakavellas SI, Tentolouris A, et al. Liver cirrhosis-effect on QT interval and cardiac autonomic nervous system activity. World J Gastrointest Pathophysiol. 2018;9(1):28-36doi: 10.4291/wjgp.v9.i1.28.
Tsiompanidis, E., Siakavellas, S. I., Tentolouris, A., Eleftheriadou, I., Chorepsima, S., Manolakis, A., Oikonomou, K., & Tentolouris, N. (2018). Liver cirrhosis-effect on QT interval and cardiac autonomic nervous system activity. World journal of gastrointestinal pathophysiology, 9(1), 28-36. https://doi.org/10.4291/wjgp.v9.i1.28
Tsiompanidis, Elias, et al. "Liver cirrhosis-effect on QT interval and cardiac autonomic nervous system activity." World journal of gastrointestinal pathophysiology vol. 9,1 (2018): 28-36. doi: https://doi.org/10.4291/wjgp.v9.i1.28
Tsiompanidis E, Siakavellas SI, Tentolouris A, Eleftheriadou I, Chorepsima S, Manolakis A, Oikonomou K, Tentolouris N. Liver cirrhosis-effect on QT interval and cardiac autonomic nervous system activity. World J Gastrointest Pathophysiol. 2018 Feb 15;9(1):28-36. doi: 10.4291/wjgp.v9.i1.28. PMID: 29487764; PMCID: PMC5823700.
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Nonalcoholic fatty liver disease: Diagnostic biomarkers.

World journal of gastrointestinal pathophysiology

Hadizadeh F, Faghihimani E, Adibi P.
PMID: 28573064
World J Gastrointest Pathophysiol. 2017 May 15;8(2):11-26. doi: 10.4291/wjgp.v8.i2.11.

Nonalcoholic fatty liver disease is a common medical condition worldwide and its prevalence has increased notably in the past few years due to the increases in prevalence of obesity and metabolic syndrome. However, diagnosis of this disease is still...

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Hadizadeh F, Faghihimani E, Adibi P. Nonalcoholic fatty liver disease: Diagnostic biomarkers. World J Gastrointest Pathophysiol. 2017;8(2):11-26doi: 10.4291/wjgp.v8.i2.11.
Hadizadeh, F., Faghihimani, E., & Adibi, P. (2017). Nonalcoholic fatty liver disease: Diagnostic biomarkers. World journal of gastrointestinal pathophysiology, 8(2), 11-26. https://doi.org/10.4291/wjgp.v8.i2.11
Hadizadeh, Fatemeh, et al. "Nonalcoholic fatty liver disease: Diagnostic biomarkers." World journal of gastrointestinal pathophysiology vol. 8,2 (2017): 11-26. doi: https://doi.org/10.4291/wjgp.v8.i2.11
Hadizadeh F, Faghihimani E, Adibi P. Nonalcoholic fatty liver disease: Diagnostic biomarkers. World J Gastrointest Pathophysiol. 2017 May 15;8(2):11-26. doi: 10.4291/wjgp.v8.i2.11. PMID: 28573064; PMCID: PMC5437499.
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Late onset pulmonary metastasis more than 10 years after primary sigmoid carcinoma.

World journal of gastrointestinal pathophysiology

Daniels AM, Vogelaar JFJ.
PMID: 28573073
World J Gastrointest Pathophysiol. 2017 May 15;8(2):96-99. doi: 10.4291/wjgp.v8.i2.96.

According to current guidelines, follow-up of patients with colorectal cancer is ended after five years. Also, chest X-ray is not part of standard investigation during follow-up. We describe a case of a 74-year-old patient, more than ten years after...

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Daniels AM, Vogelaar JFJ. Late onset pulmonary metastasis more than 10 years after primary sigmoid carcinoma. World J Gastrointest Pathophysiol. 2017;8(2):96-99doi: 10.4291/wjgp.v8.i2.96.
Daniels, A. M., & Vogelaar, J. F. J. (2017). Late onset pulmonary metastasis more than 10 years after primary sigmoid carcinoma. World journal of gastrointestinal pathophysiology, 8(2), 96-99. https://doi.org/10.4291/wjgp.v8.i2.96
Daniels, Anne M, and Vogelaar, Jeroen F J. "Late onset pulmonary metastasis more than 10 years after primary sigmoid carcinoma." World journal of gastrointestinal pathophysiology vol. 8,2 (2017): 96-99. doi: https://doi.org/10.4291/wjgp.v8.i2.96
Daniels AM, Vogelaar JFJ. Late onset pulmonary metastasis more than 10 years after primary sigmoid carcinoma. World J Gastrointest Pathophysiol. 2017 May 15;8(2):96-99. doi: 10.4291/wjgp.v8.i2.96. PMID: 28573073; PMCID: PMC5437508.
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Emergency resection surgery for colorectal cancer: Patterns of recurrent disease and survival.

World journal of gastrointestinal pathophysiology

Littlechild J, Junejo M, Simons AM, Curran F, Subar D.
PMID: 29487762
World J Gastrointest Pathophysiol. 2018 Feb 15;9(1):8-17. doi: 10.4291/wjgp.v9.i1.8.

AIM: To evaluate prognostic pathological factors associated with early metachronous disease and adverse long-term survival in these patients.METHODS: Clinical and histological features were analysed retrospectively over an eight-year period for prognostic impact on recurrent disease and overall survival in...

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Littlechild J, Junejo M, Simons AM, et al. Emergency resection surgery for colorectal cancer: Patterns of recurrent disease and survival. World J Gastrointest Pathophysiol. 2018;9(1):8-17doi: 10.4291/wjgp.v9.i1.8.
Littlechild, J., Junejo, M., Simons, A. M., Curran, F., & Subar, D. (2018). Emergency resection surgery for colorectal cancer: Patterns of recurrent disease and survival. World journal of gastrointestinal pathophysiology, 9(1), 8-17. https://doi.org/10.4291/wjgp.v9.i1.8
Littlechild, Joe, et al. "Emergency resection surgery for colorectal cancer: Patterns of recurrent disease and survival." World journal of gastrointestinal pathophysiology vol. 9,1 (2018): 8-17. doi: https://doi.org/10.4291/wjgp.v9.i1.8
Littlechild J, Junejo M, Simons AM, Curran F, Subar D. Emergency resection surgery for colorectal cancer: Patterns of recurrent disease and survival. World J Gastrointest Pathophysiol. 2018 Feb 15;9(1):8-17. doi: 10.4291/wjgp.v9.i1.8. PMID: 29487762; PMCID: PMC5823701.
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Differential role of Hedgehog signaling in human pancreatic (patho-) physiology: An up to date review.

World journal of gastrointestinal pathophysiology

Klieser E, Swierczynski S, Mayr C, Jäger T, Schmidt J, Neureiter D, Kiesslich T, Illig R.
PMID: 27190692
World J Gastrointest Pathophysiol. 2016 May 15;7(2):199-210. doi: 10.4291/wjgp.v7.i2.199.

Since the discovery of the Hedgehog (Hh) pathway in drosophila melanogaster, our knowledge of the role of Hh in embryonic development, inflammation, and cancerogenesis in humans has dramatically increased over the last decades. This is the case especially concerning...

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Klieser E, Swierczynski S, Mayr C, et al. Differential role of Hedgehog signaling in human pancreatic (patho-) physiology: An up to date review. World J Gastrointest Pathophysiol. 2016;7(2):199-210doi: 10.4291/wjgp.v7.i2.199.
Klieser, E., Swierczynski, S., Mayr, C., Jäger, T., Schmidt, J., Neureiter, D., Kiesslich, T., & Illig, R. (2016). Differential role of Hedgehog signaling in human pancreatic (patho-) physiology: An up to date review. World journal of gastrointestinal pathophysiology, 7(2), 199-210. https://doi.org/10.4291/wjgp.v7.i2.199
Klieser, Eckhard, et al. "Differential role of Hedgehog signaling in human pancreatic (patho-) physiology: An up to date review." World journal of gastrointestinal pathophysiology vol. 7,2 (2016): 199-210. doi: https://doi.org/10.4291/wjgp.v7.i2.199
Klieser E, Swierczynski S, Mayr C, Jäger T, Schmidt J, Neureiter D, Kiesslich T, Illig R. Differential role of Hedgehog signaling in human pancreatic (patho-) physiology: An up to date review. World J Gastrointest Pathophysiol. 2016 May 15;7(2):199-210. doi: 10.4291/wjgp.v7.i2.199. PMID: 27190692; PMCID: PMC4867399.
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Current therapy of pediatric Crohn's disease.

World journal of gastrointestinal pathophysiology

Lahad A, Weiss B.
PMID: 25977836
World J Gastrointest Pathophysiol. 2015 May 15;6(2):33-42. doi: 10.4291/wjgp.v6.i2.33.

Inflammatory bowel diseases (IBD), including Crohn's disease (CD) and ulcerative colitis, are chronic relapsing and remitting diseases of the bowel, with an unknown etiology and appear to involve interaction between genetic susceptibility, environmental factors and the immune system. Although...

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Lahad A, Weiss B. Current therapy of pediatric Crohn's disease. World J Gastrointest Pathophysiol. 2015;6(2):33-42doi: 10.4291/wjgp.v6.i2.33.
Lahad, A., & Weiss, B. (2015). Current therapy of pediatric Crohn's disease. World journal of gastrointestinal pathophysiology, 6(2), 33-42. https://doi.org/10.4291/wjgp.v6.i2.33
Lahad, Avishay, and Weiss, Batia. "Current therapy of pediatric Crohn's disease." World journal of gastrointestinal pathophysiology vol. 6,2 (2015): 33-42. doi: https://doi.org/10.4291/wjgp.v6.i2.33
Lahad A, Weiss B. Current therapy of pediatric Crohn's disease. World J Gastrointest Pathophysiol. 2015 May 15;6(2):33-42. doi: 10.4291/wjgp.v6.i2.33. PMID: 25977836; PMCID: PMC4419092.
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Lymphoproliferative disorders in inflammatory bowel disease patients on immunosuppression: Lessons from other inflammatory disorders.

World journal of gastrointestinal pathophysiology

Lam GY, Halloran BP, Peters AC, Fedorak RN.
PMID: 26600976
World J Gastrointest Pathophysiol. 2015 Nov 15;6(4):181-92. doi: 10.4291/wjgp.v6.i4.181.

Immunosuppressive agents, such as thiopurines, methotrexate, and biologics, have revolutionized the treatment of inflammatory bowel disease (IBD). However, a number of case reports, case control studies and retrospective studies over the last decade have identified a concerning link between...

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Lam GY, Halloran BP, Peters AC, et al. Lymphoproliferative disorders in inflammatory bowel disease patients on immunosuppression: Lessons from other inflammatory disorders. World J Gastrointest Pathophysiol. 2015;6(4):181-92doi: 10.4291/wjgp.v6.i4.181.
Lam, G. Y., Halloran, B. P., Peters, A. C., & Fedorak, R. N. (2015). Lymphoproliferative disorders in inflammatory bowel disease patients on immunosuppression: Lessons from other inflammatory disorders. World journal of gastrointestinal pathophysiology, 6(4), 181-92. https://doi.org/10.4291/wjgp.v6.i4.181
Lam, Grace Y, et al. "Lymphoproliferative disorders in inflammatory bowel disease patients on immunosuppression: Lessons from other inflammatory disorders." World journal of gastrointestinal pathophysiology vol. 6,4 (2015): 181-92. doi: https://doi.org/10.4291/wjgp.v6.i4.181
Lam GY, Halloran BP, Peters AC, Fedorak RN. Lymphoproliferative disorders in inflammatory bowel disease patients on immunosuppression: Lessons from other inflammatory disorders. World J Gastrointest Pathophysiol. 2015 Nov 15;6(4):181-92. doi: 10.4291/wjgp.v6.i4.181. PMID: 26600976; PMCID: PMC4644882.
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Role of cancer stem cells in age-related rise in colorectal cancer.

World journal of gastrointestinal pathophysiology

Nangia-Makker P, Yu Y, Majumdar AP.
PMID: 26600965
World J Gastrointest Pathophysiol. 2015 Nov 15;6(4):86-9. doi: 10.4291/wjgp.v6.i4.86.

Colorectal cancer (CRC) that comprises about 50% of estimated gastrointestinal cancers remains a high mortality malignancy. It is estimated that CRC will result in 9% of all cancer related deaths. CRC is the third leading malignancy affecting both males...

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Nangia-Makker P, Yu Y, Majumdar AP. Role of cancer stem cells in age-related rise in colorectal cancer. World J Gastrointest Pathophysiol. 2015;6(4):86-9doi: 10.4291/wjgp.v6.i4.86.
Nangia-Makker, P., Yu, Y., & Majumdar, A. P. (2015). Role of cancer stem cells in age-related rise in colorectal cancer. World journal of gastrointestinal pathophysiology, 6(4), 86-9. https://doi.org/10.4291/wjgp.v6.i4.86
Nangia-Makker, Pratima, et al. "Role of cancer stem cells in age-related rise in colorectal cancer." World journal of gastrointestinal pathophysiology vol. 6,4 (2015): 86-9. doi: https://doi.org/10.4291/wjgp.v6.i4.86
Nangia-Makker P, Yu Y, Majumdar AP. Role of cancer stem cells in age-related rise in colorectal cancer. World J Gastrointest Pathophysiol. 2015 Nov 15;6(4):86-9. doi: 10.4291/wjgp.v6.i4.86. PMID: 26600965; PMCID: PMC4644890.
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Alcoholic hepatitis: The pivotal role of Kupffer cells.

World journal of gastrointestinal pathophysiology

Suraweera DB, Weeratunga AN, Hu RW, Pandol SJ, Hu R.
PMID: 26600966
World J Gastrointest Pathophysiol. 2015 Nov 15;6(4):90-8. doi: 10.4291/wjgp.v6.i4.90.

Kupffer cells play a central role in the pathogenesis of alcoholic hepatitis (AH). It is believed that alcohol increases the gut permeability that results in raised levels of serum endotoxins containing lipopolysaccharides (LPS). LPS binds to LPS-binding proteins and...

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Suraweera DB, Weeratunga AN, Hu RW, et al. Alcoholic hepatitis: The pivotal role of Kupffer cells. World J Gastrointest Pathophysiol. 2015;6(4):90-8doi: 10.4291/wjgp.v6.i4.90.
Suraweera, D. B., Weeratunga, A. N., Hu, R. W., Pandol, S. J., & Hu, R. (2015). Alcoholic hepatitis: The pivotal role of Kupffer cells. World journal of gastrointestinal pathophysiology, 6(4), 90-8. https://doi.org/10.4291/wjgp.v6.i4.90
Suraweera, Duminda B, et al. "Alcoholic hepatitis: The pivotal role of Kupffer cells." World journal of gastrointestinal pathophysiology vol. 6,4 (2015): 90-8. doi: https://doi.org/10.4291/wjgp.v6.i4.90
Suraweera DB, Weeratunga AN, Hu RW, Pandol SJ, Hu R. Alcoholic hepatitis: The pivotal role of Kupffer cells. World J Gastrointest Pathophysiol. 2015 Nov 15;6(4):90-8. doi: 10.4291/wjgp.v6.i4.90. PMID: 26600966; PMCID: PMC4644891.
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Starring role of toll-like receptor-4 activation in the gut-liver axis.

World journal of gastrointestinal pathophysiology

Carotti S, Guarino MP, Vespasiani-Gentilucci U, Morini S.
PMID: 26600967
World J Gastrointest Pathophysiol. 2015 Nov 15;6(4):99-109. doi: 10.4291/wjgp.v6.i4.99.

Since the introduction of the term "gut-liver axis", many studies have focused on the functional links of intestinal microbiota, barrier function and immune responses to liver physiology. Intestinal and extra-intestinal diseases alter microbiota composition and lead to dysbiosis, which...

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Carotti S, Guarino MP, Vespasiani-Gentilucci U, et al. Starring role of toll-like receptor-4 activation in the gut-liver axis. World J Gastrointest Pathophysiol. 2015;6(4):99-109doi: 10.4291/wjgp.v6.i4.99.
Carotti, S., Guarino, M. P., Vespasiani-Gentilucci, U., & Morini, S. (2015). Starring role of toll-like receptor-4 activation in the gut-liver axis. World journal of gastrointestinal pathophysiology, 6(4), 99-109. https://doi.org/10.4291/wjgp.v6.i4.99
Carotti, Simone, et al. "Starring role of toll-like receptor-4 activation in the gut-liver axis." World journal of gastrointestinal pathophysiology vol. 6,4 (2015): 99-109. doi: https://doi.org/10.4291/wjgp.v6.i4.99
Carotti S, Guarino MP, Vespasiani-Gentilucci U, Morini S. Starring role of toll-like receptor-4 activation in the gut-liver axis. World J Gastrointest Pathophysiol. 2015 Nov 15;6(4):99-109. doi: 10.4291/wjgp.v6.i4.99. PMID: 26600967; PMCID: PMC4644892.
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Host-microbiome interaction in Crohn's disease: A familiar or familial issue?.

World journal of gastrointestinal pathophysiology

Michielan A, D'Incà R.
PMID: 26600974
World J Gastrointest Pathophysiol. 2015 Nov 15;6(4):159-68. doi: 10.4291/wjgp.v6.i4.159.

An impaired interaction between the gut and the intestinal microbiome is likely to be the key element in the pathogenesis of Crohn's disease (CD). Family studies have provided invaluable information on CD pathogenesis and on its etiology. Relatives share...

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Michielan A, D'Incà R. Host-microbiome interaction in Crohn's disease: A familiar or familial issue?. World J Gastrointest Pathophysiol. 2015;6(4):159-68doi: 10.4291/wjgp.v6.i4.159.
Michielan, A., & D'Incà, R. (2015). Host-microbiome interaction in Crohn's disease: A familiar or familial issue?. World journal of gastrointestinal pathophysiology, 6(4), 159-68. https://doi.org/10.4291/wjgp.v6.i4.159
Michielan, Andrea, and D'Incà, Renata. "Host-microbiome interaction in Crohn's disease: A familiar or familial issue?." World journal of gastrointestinal pathophysiology vol. 6,4 (2015): 159-68. doi: https://doi.org/10.4291/wjgp.v6.i4.159
Michielan A, D'Incà R. Host-microbiome interaction in Crohn's disease: A familiar or familial issue?. World J Gastrointest Pathophysiol. 2015 Nov 15;6(4):159-68. doi: 10.4291/wjgp.v6.i4.159. PMID: 26600974; PMCID: PMC4644880.
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Faecal calprotectin: Management in inflammatory bowel disease.

World journal of gastrointestinal pathophysiology

Benítez JM, García-Sánchez V.
PMID: 26600978
World J Gastrointest Pathophysiol. 2015 Nov 15;6(4):203-9. doi: 10.4291/wjgp.v6.i4.203.

Inflammatory bowel disease (IBD) is a chronic and relapsing disorder which leads to an inflammation of the gastrointestinal tract. A tailored therapy to achieve mucosal healing with the less adverse events has become a key issue in the management...

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Benítez JM, García-Sánchez V. Faecal calprotectin: Management in inflammatory bowel disease. World J Gastrointest Pathophysiol. 2015;6(4):203-9doi: 10.4291/wjgp.v6.i4.203.
Benítez, J. M., & García-Sánchez, V. (2015). Faecal calprotectin: Management in inflammatory bowel disease. World journal of gastrointestinal pathophysiology, 6(4), 203-9. https://doi.org/10.4291/wjgp.v6.i4.203
Benítez, José Manuel, and García-Sánchez, Valle. "Faecal calprotectin: Management in inflammatory bowel disease." World journal of gastrointestinal pathophysiology vol. 6,4 (2015): 203-9. doi: https://doi.org/10.4291/wjgp.v6.i4.203
Benítez JM, García-Sánchez V. Faecal calprotectin: Management in inflammatory bowel disease. World J Gastrointest Pathophysiol. 2015 Nov 15;6(4):203-9. doi: 10.4291/wjgp.v6.i4.203. PMID: 26600978; PMCID: PMC4644884.
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Showing 1 to 12 of 258 entries